SMASH, a fragmentation and sequencing method for genomic copy number analysis.
Genetics
A rapid chemical-genetic screen utilizing impaired movement phenotypes in C. elegans: Input into genetics of neurodevelopmental disorders.
Hyperactivity and male-specific sleep deficits in the 16p11.2 deletion mouse model of autism.
Strong association of de novo copy number mutations with autism.
Brain evolution and uniqueness in the human genome.
High-risk autism spectrum disorder Utah pedigrees: a novel shared genomic segments analysis.
Unusually rapid evolution of neuroligin-4 in mice.
Identifying autism loci and genes by tracing recent shared ancestry.
Failure of neuronal homeostasis results in common neuropsychiatric phenotypes.
Association between microdeletion and microduplication at 16p11.2 and autism.
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