16p11.2 deletion syndrome mice display sensory and ultrasonic vocalization deficits during social interactions.
Dynamics of BAF-Polycomb complex opposition on heterochromatin in normal and oncogenic states.
Clustering autism: Using neuroanatomical differences in 26 mouse models to gain insight into the heterogeneity.
The autism inpatient collection: Methods and preliminary sample description.
In tribute to Bob Blanchard: Divergent behavioral phenotypes of 16p11.2 deletion mice reared in same-genotype versus mixed-genotype cages.
16p11.2 deletion mice display cognitive deficits in touchscreen learning and novelty recognition tasks.
Behavioral abnormalities and circuit defects in the basal ganglia of a mouse model of 16p11.2 deletion syndrome.
Communication impairments in mice lacking Shank1: reduced levels of ultrasonic vocalizations and scent marking behavior.
Haploinsufficiency of the autism-associated SHANK3 gene leads to deficits in synaptic function, social interaction, and social communication.
R-baclofen reverses cognitive deficits and improves social interactions in two lines of 16p11.2 deletion mice.
- Previous Page
- Viewing
- Next Page