Nucleosome turnover regulates histone methylation patterns over the genome.
Loss of the neural-specific BAF subunit ACTL6B relieves repression of early response genes and causes recessive autism.
BAF subunit switching regulates chromatin accessibility to control cell cycle exit in the developing mammalian cortex.
Systemic enhancement of serotonin signaling reverses social deficits in multiple mouse models for ASD.
GABAB receptor modulation of visual sensory processing in adults with and without autism spectrum disorder.
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