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  • Tuberous sclerosis, fragile X may be molecular opposites
    1 December 2011
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    Some forms of autism are caused by too many proteins at the synapse, the junction between neurons, whereas other forms result from too few, according to a study published 23 November in Nature.

  • Molecular mechanisms: SHANK2 mutants alter synapses
    30 November 2011
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    Three mutations in SHANK2, an autism-associated gene, each lead to abnormal synapses, the junctions between neurons, according to a study in Human Molecular Genetics.

  • New genetic approach identifies autism candidate genes
    30 November 2011
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    Regions of the genome that contain the same genetic variants on both copies of a chromosome are more likely to contain autism-associated genes than other parts of the genome, according to a study published 14 October in Human Genetics.

  • Fishy findings
    29 November 2011
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    Supplements of omega-3 fatty acids, prime ingredients of fish oils, don't improve symptoms in children with autism, according to a new review.

  • Cognition and behavior: Some neurons attuned to whole faces
    29 November 2011
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    A subset of neurons in the amygdala, a small brain region responsible for processing emotions, recognize whole faces rather than individual features, according to a study published 11 October in Current Biology.